bioRxiv Science⌕ Search

bioRxiv · 10.64898/2026.11.04.764164

Multi-objective domain adaptation for pan-cancer transfer of pharmacogenomic drug response to patient tumours

Abstract

Large-scale pharmacogenomic screens provide rich drug-sensitivity measurements in cancer cell lines, but transferring these measurements to patient tumours remains limited by biological heterogeneity and cross-domain distribution shift. Existing machine learning approaches, including adversarial autoencoder and domain adaptation, address this gap by learning shared latent representations between preclinical models and patient samples. However, alignment without explicit biological constraints can produce unreliable representations when cancer lineage structure and clinically relevant patient heterogeneity are not preserved. Here, we introduce MODAE+, a pan-cancer multi-objective deep learning domain adaptation framework that jointly balances domain alignment, deconfounding, drug-response prediction, survival prediction, and cancer-lineage classification across 9,696 TCGA tumours, 1,293 CCLE cancer cell lines, and CTRP sensitivity profiles for 544 compounds in 738 matched cell lines. This multi-objective design improved cross-domain alignment while preserving cancer-lineage organization and patient heterogeneity. For drugs shared between the pharmacogenomic screen and TCGA treatment records, MODAE+ predicted sensitivity was significantly associated with overall survival among patients who received each drug in 46.3% of evaluated cancer-drug pairs by log-rank testing and 41.5% by multivariable Cox analysis. Biological validation across four curated drug-gene resources identified significant expression differences exceeding 1.5-fold for 34.9-64.4% of drugs in pan-cancer analyses, based on the lowest-p-value annotated gene per drug. Drug-class-specific patterns involved intended molecular targets for targeted therapies and predominantly transport, metabolism, detoxification and resistance-related genes for classical chemotherapies. As an additional validation, we derived pretreatment responder-nonresponder gene-expression signatures from 13 external breast cancer datasets and evaluated their concordance with MODAE+ predictions across 69 signature-drug comparisons in TCGA patients treated with paclitaxel or doxorubicin. Consensus signature scores showed Spearman correlations of 0.791 with predicted paclitaxel sensitivity and 0.724 with predicted doxorubicin sensitivity, averaging 0.758 across the two drugs. Together, these complementary analyses provide supporting evidence for the clinical relevance and biological consistency of MODAE+ predictions, suggesting their potential to identify drug-sensitive tumour subgroups and generate hypotheses about mechanisms of drug resistance.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

Fortino, V., Rintala, T. J.. 2026-11-05. Multi-objective domain adaptation for pan-cancer transfer of pharmacogenomic drug response to patient tumours. https://doi.org/10.64898/2026.11.04.764164

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related preprints

Accelerating discovery and integration from RNA to Protein with R2T2P

Integrating transcriptomics and proteomics data remains an unmet challenge in the post-genomics era, discouraging researchers from pursuing integrative approaches to molecular biology research. Leveraging the power of translation profiling, we present RNA to Translation to Protein (R2T2P), a computational platform integrating genome-guided transcriptome assembly with identification of translation and proteins, including a tailored differential analysis framework and support for multiple reference transcriptomes. We applied R2T2P to investigate a cellular model of facioscapulohumeral muscular dystrophy (FSHD), where ectopic expression of the DUX4 retrogene in skeletal muscle triggers significant alterations in transcription and RNA metabolism. We uncovered hundreds of uncharacterized, highly overexpressed RNAs, including antisense and alternative transcripts, and quantified their contribution to translatome and proteome. Re-analysis of expression datasets from patient-derived myotubes and muscle biopsies validated this alternative transcriptome in FSHD patients, adding a reservoir of bona-fide novel molecular actors in a rare disease. Available at https://github.com/nfdata-omics/R2T2P/, R2T2P is a flexible and powerful workflow in modern integrative biology.

bioinformatics↗

MorphoANOVA: Dissecting Histology-Gene Expression Relationships in Spatial Transcriptomics

Spatial transcriptomics (ST) measures the expression of hundreds to thousands of genes within tissue architecture, often accompanied by histology images. However, it remains challenging to systematically characterize which genes exhibit associations with tissue morphology, whether these associations vary spatially, and which genes show additional spatial expression patterns beyond those captured by morphology. Here we introduce MorphoANOVA, a gene-wise statistical framework for characterizing morphology--expression relationships across genes measured by ST. Using nested spatially varying coefficient models and sequential ANOVA, MorphoANOVA assesses slide-wide associations with an H\&E-derived morphology feature, location-specific changes in these associations, and additional spatial variation after accounting for the selected feature. The resulting statistics enable gene prioritization and pathway enrichment analysis, while spatial component maps support the interpretation of individual genes. Simulations demonstrate MorphoANOVA's ability to distinguish the modeled components and recover their spatial patterns. Applications to human cortex and breast cancer identify biological programs associated with different morphology--expression patterns and reveal distinct relationships with cell composition. Across three HER2-positive breast cancer patients, invasive-cancer marker genes consistently exhibit stronger evidence of location-specific morphology associations. MorphoANOVA thus provides a framework for systematically interpreting spatial gene-expression patterns in relation to tissue morphology.

bioinformatics↗

Evaluation effects can rival reported concordance gains in cross-cancer survival prediction

Studies of cross-cancer learning for survival prediction from tumour omics have reported concordance gains of 0.020 and 0.045, but how such gains compare with evaluation effects remains unclear. We examined these effects across 13 TCGA cancers (5,761 patients, 1,526 disease-specific deaths), using five repeats of five-fold cross-validation and paired bootstrap intervals. When model selection did not use test data, our borrowing model gained 0.009 (95% interval 0.001 to 0.016) over its target-fitted reference when cancers received equal weight (post hoc). Random-effects pooling gave a gain of 0.002 (-0.002 to 0.006), with most weight assigned to cancers where the model made little use of the pretrained score. Choosing the fusion weight on test splits raised the apparent equal-weight gain to 0.017. We reimplemented a published method, VAECox, which showed no pretraining gain over random initialisation at the last or validation-selected training step. Choosing each network's best test step, as the published code does, raised concordance by 0.03 to 0.05 across the 13-cancer and ten-cancer overall-survival settings, but barely changed the difference between networks. In a constructed post hoc experiment, choosing the best test step only for the pretrained network changed the equal-weight mean difference from -0.004 to +0.045 (+0.035 to +0.054). Across seven multi-omics comparisons, estimates from single five-fold runs differed from their cancer's five-run mean by at least 0.02 in 11% to 42% of runs. Concordance was about 0.02 lower at held-out tissue source sites. Interpreting a reported gain therefore requires each model's selection rule, variation across repeated partitions, the weighting of cancers and the evaluation population.

bioinformatics↗