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bioRxiv · 10.64898/2026.08.28.747755

De novo design of CR2 binder as vaccine scaffold

Abstract

Efficient B cell activation during vaccine-induced humoral immunity relies on both B cell receptor (BCR) antigen recognition and synergistic signaling from co-receptors. Complement receptor 2 (CR2), the primary BCR co-receptor on B cells, lowers the activation threshold and amplifies downstream kinase signaling by orders of magnitude when engaged by complement fragment C3d decorated antigens. Targeting CR2 therefore represents a rational vaccine enhancement strategy, yet native C3d suffers from low affinity, poor stability, and manufacturing challenges. Here, we report the de novo design of a highly stable, high-affinity CR2 binder using deep learning driving protein design methods. Biophysical characterization, high-resolution cryoEM structural determination, and functional assays in vitro and in vivo confirm that the designed binder matches computational design models and specifically engages CR2 to boost B cell activation. When fused to antigen as a vaccine scaffold, the trimeric CR2 binder elicits robust humoral immune responses comparable to nanoparticle vaccines, while retaining the simplicity of single-chain protein production. Our work establishes a modular CR2 targeting vaccine scaffold platform with broad translational potential for next-generation protein vaccines.

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Liu, Y.-T., Yin, Y.-N., Xu, H.-Q., Du, W.-T., Xie, C., Wu, P.-H., Zhou, H., Cheng, B.-Z., Bu, G.-L., Feng, G.-K., Zhong, Q., Liu, Z., Zeng, M.-S., Sun, C.. 2026-09-03. De novo design of CR2 binder as vaccine scaffold. https://doi.org/10.64898/2026.08.28.747755

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