bioRxiv Science⌕ Search

bioRxiv · 10.1101/2025.11.03.686224

M4 drug discovery: human drug predictions from integrated pre-clinical insights exemplified with a GLP1-R agonist

Abstract

A major recent breakthrough in the treatment of type 2 diabetes has been the development of glucagon-like peptide-1 receptor agonists (GLP-1RAs). However, current translational frameworks struggle to predict the clinical outcomes of these drugs from preclinical data. There are several reasons for this struggle, which are generic for many drugs: GLP-1RAs act through multi-timescale mechanisms in which short-term effects propagate into long-term changes; no single preclinical system can capture all their effects in humans; and mechanistic extrapolation requires modelling numerous whole-body biological processes. To address this gap, we present a new extrapolation approach, M4 drug discovery, and retrospectively apply it to the GLP-1RA exenatide in a manner that is generalisable to other drugs. The method integrates: Multi-level data (cellular to whole-body), Multi-timescale data (minutes to months), Multi-species data (e.g., rodents to humans), and Mechanistic knowledge. In this study, we integrate human cell and animal data with drug-free human studies to successfully predict human pharmacokinetics (cost < {chi}2, p=0.05; 64 < 97) and the outcomes of a 30-week clinical trial (36 < 45). We found that integrating information across the four M4 axes improved predictive performance and physiological relevance: multi-species data inform pharmacokinetics, human cell data provide human population- and donor-specific potency estimates, animal data reveal additional drug effects not observable in cell cultures, and the multi-timescale mathematical modelling enables short-term effects of exenatide and meals to inform long-term changes in insulin sensitivity. This work provides new tools for drug extrapolations, supporting the community towards safer and more informed preclinical-to-clinical drug extrapolations.

Source connections

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

Silfvergren, O., Rigal, S., Schimek, K., Simonsson, C., Kanebratt, K. P., Forschler, F., Yesildag, B., Marx, U., Vilen, L., Gennemark, P., Cedersund, G.. 2025-11-05. M4 drug discovery: human drug predictions from integrated pre-clinical insights exemplified with a GLP1-R agonist. https://doi.org/10.1101/2025.11.03.686224

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related preprints

Large language models complement established workflows for mechanistic model construction and refinement in systems biology

Mechanistic models provide a framework for describing, understanding, and predicting biological processes, but their development requires an interdisciplinary team with expertise in biology, mathematics, and computation. Here we present Octomod, a multi-agent workflow based on large language models (LLMs) that integrates biological knowledge retrieval, model construction, parameter estimation, and hypothesis testing. Built on established software tools, including AMICI and pyPESTO, and community standards, including SBML and PEtab, Octomod supports model development and refinement through natural-language interaction with a user. The agents' proposals must pass defined checks and the user's decision before they are used. Every input, model, check, and decision is recorded, so that model building remains transparent and reproducible. We applied Octomod to four problems. Three used published data and differ in model size and in how much the user guided the run: Octomod constructed a model that matched a published model without user guidance, supported the interactive refinement of a gene-regulatory model, and built models of cell-cell communication that explained the responses of five immune cell types to different stimuli. In the fourth application, on newly generated and unpublished lipidomics data, Octomod helped to determine which processes the measurements could inform, identify unresolved label fluxes, and reduce a supplied model to those processes. Together, these results show how LLMs can strengthen and complement established workflows for constructing and refining biologically informed models.

systems biology↗

A Mathematical Model Connecting Appetite-Driven Food Intake to Metabolic Changes across Diet and GLP-1RA Treatments in Mice

Mouse models are widely used to study obesity and the effects of glucagon-like peptide-1 receptor agonists (GLP-1RAs) on appetite-driven meal responses and eating behaviours. However, connecting these behavioural changes to disease prevention remains limited. One reason is that eating behaviour is often summarised as daily or weekly energy intake, obscuring meal-level dynamics. Here we present a mathematical model of mouse appetite-driven food intake across diets and treatment with the GLP-1RAs semaglutide and exenatide. The model jointly described three studies and passed both qualitative assessments and a statistical {chi}2 test (disagreement<threshold, 348<1111). Predictive performance was subsequently validated using four pre-existing studies and one new study. Finally, the model was used to simulate mouse populations under dietary and GLP-1RA interventions, with predictions showing qualitative agreement with the literature. This work aids preclinical drug discovery, supports mouse study design, and improves understanding of how individual meals accumulate into long-term physiological changes.

systems biology↗

Generalized inference of noise-corrected state variability in single cells

Single-cell omics methods operate at their detection limit and are less conducive to technical controls than mini-bulk approaches, which combine cells of the same type before profiling. To integrate mini-bulk samples with paired split-pool controls more holistically, we built stochprofIC, an algorithm that considers data, noise, and covariates in a distributional-regression framework. We vet stochprofIC with synthetic datasets and compare against an adjusted-variance alternative using mini-bulk data in mouse brains and human breast cancers. The noise-batch corrections provided by stochprofIC enable inference of the single-cell populations comprising the mini-bulk, which are validated repeatedly by RNA FISH in different contexts. We use stochprofIC outputs as input to a systems model of nucleocytoplasmic transport and distinguish heterogeneous trafficking states. Pharmacologic inhibition of a MAP3K11-high state inhibits apoptosis in primary breast-cancer organoids, supporting relevance to phenotypes. By deconvolving changes in location and scale, stochprofIC supports mini-bulking when high-sensitivity and reliability profiles are important.

systems biology↗