bioRxiv Science⌕ Search

bioRxiv · 10.1101/2025.06.28.662110

Structural Basis for Efficient Fo Motor Rotation Revealed by MCMD simulation and Structural Analysis

Abstract

Fo domain of ATP synthase functions as a rotary molecular motor, coupling proton translocation with the rotation of the c-ring rotor. This process involves proton uptake at the entry half channel, rotor rotation, and proton release to the exit half channel. While the overall coupling mechanism is established, the design principle for efficient rotation remains unclear. Here, we employed hybrid molecular simulations--combining coarse-grained modeling and Monte Carlo methods--to investigate the roles of side chain flexibility at proton-binding residues and the angular mismatch between the proton uptake process and the proton release process. Our results indicate that both factors promote rotational activity, with side chain flexibility playing a more significant role. Comparable analysis of Fo structures from different species revealed that the key residue geometry is conserved, and that the asymmetric geometry of the two half channels aligns with the mechanism suggested by simulation. These findings highlight a conserved design principle that enhances rotational efficiency and offer a mechanistic basis for engineering synthetic rotary systems. Significance StatementFo motor is the proton-conducting unit of ATP synthase and a rotary molecular motor driven by proton translocation across the membrane. Previous biochemical and structural studies have established the "half-channel model," which explains how proton translocation is coupled with rotation. However, it remains unclear what structural features of Fo enable the smooth coordination of proton flow and rotation for kinetically efficient motion. In this study, we conducted a hybrid molecular simulation combining coarse-grained model calculations with Monte Carlo simulations. The present study found that the conformational flexibility of side chains in amino acid residues involved in proton translocation is one of the critical factors for kinetically efficient rotation. Furthermore, it is also found that the fundamental structural features are conserved across different species, suggesting that the principal mechanism and the design principles of Fo motor are well conserved across species.

Source connections

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

Kubo, S., Noji, H.. 2025-07-02. Structural Basis for Efficient Fo Motor Rotation Revealed by MCMD simulation and Structural Analysis. https://doi.org/10.1101/2025.06.28.662110

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related preprints

A quantum state of mitochondria in the living cell

The high energy-efficiency of life is hard to understand only with classical physics. Many efforts have been made to study its mechanism based on quantum mechanics; the progress is nevertheless slow due to lack of experimental evidence with living cells. Here, combining experiments on cells, tissues and mitochondria with a theoretical model, we demonstrate a quantum state of mitochondria, which can be employed to modulate ATP production in living cells. We found an anomalous 71.0-THz oscillation mode only in living cells and tissues, which is highly determined by intact structure of mitochondria, and cannot be assigned to any specific molecules. Based on experimental data, a quantum model of light-matter coupling was introduced to trace the origin of this mode. Our calculations suggest a quantum superposition state of functional mitochondrion that forms by the coupling of light and lipid CH2 bonds in functional cristae, and induces a splitting of the intrinsic CH2 vibration mode of 87 THz to two levels at 71 THz and 103 THz, respectively. The former can be observed only in living cells and tissues; whereas the latter falls in the range (90-110 THz) of biomolecular and water vibrations, thus indistinguishable. Additional experiments revealed this mitochondrial quantum state able to serve as an efficient channel to modulate ATP production. Our findings provide a quantum mechanics view for understanding living cells, and it will be interesting to further explore whether such quantum state could act as a channel for energy metabolism, and even information transmission in life.

biophysics↗

Mechanistical and structural basis of Kv channel inhibition by 4 aminopyridine

Inhibition of Kv channels by 4-aminopyridine (4AP) improves motor function in multiple sclerosis by enhancing neuronal excitability. The mechanism of inhibition and the structural basis of 4AP binding to Kv channels remain unclear. Here, we determined the structure of the Shaker V369I-I372L-S376T (ILT) mutant bound to 4AP at 3.3 [A], demonstrating that 4AP binds to the closed state of the channel. This structure is inconsistent with an open channel block mechanism. Electrophysiology experiments show that 4AP binds even when intracellular pore access is constitutively blocked, suggesting that 4AP enters the pore through membrane-facing fenestrations. MD simulations and mutational analysis agree with the proposed fenestration pathway and suggest that 4AP binds in its neutral form. These results support a mechanism where 4AP binds to a partially activated closed state that prevents complete activation of Kv channels, explaining its pharmacological activity.

biophysics↗

Substrate binding reorganizes the energetic landscape of Plasmodium falciparum hexose transporter PfHT1

Malaria parasites depend on the Plasmodium falciparum hexose transporter PfHT1 for sugar uptake, yet how substrate binding reshapes transporter energetics and kinetics of sugar transport remains poorly understood. Here, we investigate how glucose reorganizes the conformational landscape, transition pathways, and residue interaction network underlying membrane transport. Using over 800 s of adaptive molecular dynamics simulations combined with Markov state models, transition-path theory, residue-contact analysis, and graph attention learning, we reconstruct the apo and glucose-bound conformational cycles. We show that glucose selectively stabilizes productive outward-facing, occluded, and inward-facing conformations, reshapes transition kinetics, and channels reactive flux through the occluded state. We identify TM7b helix cracking as a local structural transition coupled to extracellular-gate closure and substrate progression, providing a flexible connection between the binding pocket and global alternating access. Experimental testing of mechanistically critical residues validated their functional importance in PfHT1-dependent sugar utilization. Together, these results show how substrate binding reorganizes the energetic, kinetic, and interaction landscape of membrane transport and establish a transferable framework for studying transporter mechanisms.

biophysics↗