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bioRxiv · 10.1101/2024.07.17.603865

Ablation of B cell-derived IL-10 increases tuberculosis resistance

Abstract

Due to the historical dogma, that host defense against intracellular pathogens is mediated by cell-mediated immunity, B cells have been considered unimportant in providing protection against Mycobacterium tuberculosis (Mtb) and remained understudied for decades. However, emerging evidence suggests a more complex and multifaceted role for B cells in tuberculosis (TB) immunity. They accumulate at the side of infection in both animal models and human TB patients, suggesting a potential link to protective immunity. Still, the diverse roles of B cells in TB immunity continue to be unraveled. Apart from antibodies, B cells produce a wide range of cytokines, which can influence the local immune response. Here we addressed the relevance of interleukin 10 (IL-10) secreting B cells in long-term control of the Mtb Beijing strain HN878. Our research highlights the previously unknown role of B cell-derived IL-10 as a negative regulator of protective immunity in TB. For the first time, we demonstrate that mice lacking B cell-derived IL-10 show increased resistance to aerosol Mtb infection, as evidenced by a delayed onset of clinical symptoms and prolonged survival. Notably, this effect was significantly more pronounced in males compared to females, and was accompanied by male-specific immune alterations, indicating a previously unknown sex-specific regulatory role of B cell-derived IL-10 during Mtb infection.

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BibTeXRIS

Hertz, D., Marwitz, S., Eggers, L., von Borstel, L., Harikumar Parvathy, G., Behrends, J., Jonigk, D. D., Manz, R., Goldmann, T., Schneider, B. E.. 2024-07-21. Ablation of B cell-derived IL-10 increases tuberculosis resistance. https://doi.org/10.1101/2024.07.17.603865

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