bioRxiv · 10.1101/2022.06.05.494912
Induction of senescence renders cancer cells highly immunogenic
Abstract
Cellular senescence is a stress response that activates innate immunity. However, the interplay between senescent cells and the adaptive immune system remains largely unexplored. Here, we show that senescent cells display enhanced MHC class I (MHC-I) antigen processing and presentation. Immunization of mice with senescent syngeneic fibroblasts generates CD8 T cells reactive against both normal and senescent fibroblasts, some of them targeting senescence-associated MHC-I-peptides. In the context of cancer, we demonstrate that senescent cancer cells trigger strong anti-tumor protection mediated by antigen-presenting cells and CD8 T cells. This response is superior to the protection elicited by cells undergoing immunogenic cell death. Finally, induction of senescence in patient-derived cancer cells exacerbates the activation of autologous tumor-reactive CD8 tumor-infiltrating lymphocytes (TILs) with no effect on non-reactive TILs. Our study indicates that immunization with senescent cancer cells strongly activates anti-tumor immunity, and this can be exploited for cancer therapy. STATEMENT OF SIGNIFICANCEOur study shows that senescent cells are endowed with a high immunogenic potential, superior to the gold standard of immunogenic cell death. The induction of senescence in cancer cells can be exploited to develop efficient and protective CD8-dependent anti-tumor immune responses.
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Marin, I., Boix, O., Garcia-Garijo, A., Sirois, i., Caballe, A., Zarzuela, E., Ruano, I., Stephan-Otto Attolini, C., Prats, N., Lopez-Dominguez, J. A., Kovatcheva, M., Garralda, E., Munoz, J., Caron, E., Abad, M., Gros, A., Pietrocola, F., Serrano, M.. 2022-06-06. Induction of senescence renders cancer cells highly immunogenic. https://doi.org/10.1101/2022.06.05.494912
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