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bioRxiv · 10.1101/2021.09.27.461933

HIV-1 hijacks the cell extracellular matrix to spread collectively and efficiently between T lymphocytes

Abstract

Collective transmission via structures containing several virions has recently emerged as a highly efficient mode of viral spread. Here, we demonstrate that HIV-1 spreads between T lymphocytes in the form of viral particles colonies that are concentrated and sheltered in an extracellular matrix (ECM) lattice enabling their collective transmission upon cell contacts. Intrinsically, ECM-clustered viruses infect T lymphocytes more efficiently than individual viral particles. They preserve HIV-1 transmission from antiretroviral treatment (ArT) and potent broadly neutralizing antibodies. We also show that collagen induced by HIV-1 infection controls the clustering of virions and their collective spread, thereby enhancing infectivity. CD4+ T cells from HIV-1-infected patients produce and transmit ECM-virus clusters, supporting that they could be involved in vivo. This study provides new insights into modes of HIV-1 transmission and identifies a novel fundamental role for collagen in this process. HIV-1 spread via ECM-virus clusters may have important implications for viral dissemination and persistence, including during therapy.

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BibTeXRIS

Inizan, C., Caillet, M., Desrames, A., Bomme, P., MALLET, A., Bourdic, K., Lambotte, O., Boufassa, F., Mesel-Lemoine, M., Tangy, F., Pancino, G., Mouquet, H., Lagache, t., Dufour, A. C., Olivo-Marin, J.-C., Rey, F. A., Saez-Cirion, A., Thoulouze, M.-I.. 2021-09-27. HIV-1 hijacks the cell extracellular matrix to spread collectively and efficiently between T lymphocytes. https://doi.org/10.1101/2021.09.27.461933

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