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bioRxiv · 10.1101/2021.01.07.425708

Telomerase Prevents Emphysema in Old Mice by Sustaining Subpopulations of Endothelial and AT2 Cells

Abstract

Accumulation of senescent cells has been causally linked to the development of age-related pathologies. Here, we characterized a new mouse model (p21+/Tert) whose telomerase (TERT) is expressed from the p21 promoter that can be activated in response to telomere dysfunction. Lung parenchyma from p21+/Tert old mice accumulated fewer senescent cells with age and this correlated with a reduction in age-related alveolar space enlargement, a feature of pulmonary emphysema. This protection against emphysema depends on TERT catalytic activity and is associated with increased proliferation of pulmonary endothelial cells (EC) and capillary density. Single-cell RNA sequencing of lung cells revealed that TERT expression was associated with the enrichment of ECs expressing genes involved in vessel regeneration and in AT2 cells overexpressing S/G2M markers. These findings indicate that p21-promoter-dependent expression of catalytically active telomerase prevents emphysema by sustaining the proliferation of subclasses of EC and AT2 cells.

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BibTeXRIS

BREAU, M., CAYROU, C., CHURIKOV, D., FOUILLADE, C., CURRAS ALONSO, S., BAUWENS, S., JOURQUIN, F., BRAUD, L., FIORE, F., CATELLANO, R., JOSSELIN, E., SANCHEZ-FERRER, C., GIOVINAZZO, G., GILSON, E., FLORES, I., LONDONO-VALLEJO, A., ADNOT, S., GELI, V.. 2021-01-08. Telomerase Prevents Emphysema in Old Mice by Sustaining Subpopulations of Endothelial and AT2 Cells. https://doi.org/10.1101/2021.01.07.425708

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