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bioRxiv · 10.1101/2020.05.27.119198

CAR T cell entry into tumor islets is a two-step process dependent on IFNγ and ICAM-1

Abstract

Adoptive transfer of T cells expressing chimeric antigen receptors (CARs) has shown remarkable clinical efficacy against advanced B cell malignancies but not yet against solid tumors. Here, we used fluorescent imaging microscopy and ex vivo assays to compare the early functional responses (migration, Ca2+ and cytotoxicity) of CD20 and EGFR CAR T cells upon contact with malignant B and carcinoma cells. Our results indicate that CD20 CAR T cells rapidly form productive ICAM-1-dependent conjugates with their targets. By comparison, EGFR CAR T cells only interact at first with a subset of carcinoma cells located at the periphery of tumor islets. After this initial peripheral activation, EGFR CAR T cells progressively infiltrate the center of tumor cell regions. The analysis of this two-step entry process shows that activated CAR T cells trigger the upregulation of ICAM-1 on tumor cells in an IFN{gamma}-dependent pathway. The blockade of ICAM-1/LFA-1 interactions prevents CAR T cell recruitment into tumor islets. The requirement for IFN{gamma} and ICAM-1 to enable CAR T cell entry into tumor islets is of significance for improving CAR T strategy in solid tumors.

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Kantari-Mimoun, C., Barrin, S., Haghiri, S., Gervais, C., Mittelstaet, J., Mockel-Tenbrinck, N., Kinkhabwala, A., Damotte, D., Lupo, A., Sibony, M., Alifano, M., Vimeux, L., Dondi, E., Bercovici, N., Trautmann, A., Kaiser, A., Donnadieu, E.. 2020-05-30. CAR T cell entry into tumor islets is a two-step process dependent on IFNγ and ICAM-1. https://doi.org/10.1101/2020.05.27.119198

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