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da Silva, N. S.

Publications and source records attributed to da Silva, N. S..

2 recordsLinked to original sources

BIOMODULATORY EFFECT OF LOW INTENSITY LASER (830 nm.) IN NEURAL CELLS 9L/lacZ

BackgroundCurrently, research is advancing low intensity laser (LIL) in central nervous system cells to available the benefits of this therapy in neurological disorders, and research seeks to establish the best LIL protocol in biological processes of neuronal tissue with the different energy wavelengths({lambda}), and exposure time (s) and frequency(Hz). The aim of this study is to check the biomodulatory effects of the LIL in neural cell culture. MethodsDiode laser {lambda} = 830 nm, power of 40 mW, continuous mode, applied in the cells lineage 9L/lacZ, with energy densities of 0.5, 1.5 and 3.0 J/cm2. Analysed 24 hours after irradiation, the results of the cell viability show the difference between the control and treated groups. ResultsIn the occurrence of apoptosis, no significant manifestation was observed between the control group compared with the irradiated one (P = 0.9956); there was a significant difference between apoptosis and death by necrosis has been between the control and treated groups (P <0.001). In the comet assay there was not difference. ConclusionsWith the aim of evaluating whether LIL promotes early activation of programmed cell death, of 9L/lacZ cells, in the proposed parameters of LIL, we observed that promoted an increase in the number of neural cells, highlighting the action of biomodulation; LIL did not promote the activation of apoptosis and did not any indication of DNA deterioration in the comet assay. The results of this study are indicative that the near infrared laser has a positive interaction with neuronal cells.

cell biology↗

Evaluation of the effect of hydrocortisone in 2D and 3D HEp-2 cell culture

Cancer is one of the diseases with the highest incidence globally and that associated with the patients emotional state, can act positively or negatively in the treatment. Cortisol is a principal primary stress hormone in the human body. The corticoids can increase cell proliferation and reactive oxygen species that contribute to DNA damage. Prolonged exposure to stress can contribute to tissues becoming insensitive to cortisol, the primary human stress hormone. This study explores cortisols influence on tumor cell development, particularly in human cells of carcinoma of the human laryngeal (HEp-2). HEp-2 cells were exposed to increasing cortisol (hydrocortisone) concentrations for 24 or 48 hours, and cytotoxicity (MTT assay) proliferation assay (crystal violet assay), and immunolabeled 3D culture for fibronectin and FAK were analyzed. The group treated with hydrocortisone showed a significant increase in mitochondrial activity, as for the evaluation by the violet crystal, the treated group showed similar behavior to the control. The 3D culture showed dispersed cells within 24 hours with reduced FAK labeling; however, no changes were observed within 48 hours. Although some cases favored corticosteroid use in cancer patients, a more detailed analysis is necessary before prescribing them.

cancer biology↗