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Wust, A.

Publications and source records attributed to Wust, A..

2 recordsLinked to original sources

Acute threats modulate hunger circuit dynamics to instruct avoidance behaviour

During foraging animals must balance food-seeking with predator avoidance, yet how the brain integrates sensory information relating to food and threat remains unclear. Using in vivo calcium imaging in mice, we show that hunger-sensitive AgRP neurons in the hypothalamus are rapidly inhibited by threats across a threat imminence continuum, from a low environmental risk to a high physical restraint threat, independent of fasting state. This suppression is driven by GABAergic inputs from dorsomedial hypothalamus (DMH) neurons, which increase activity during threat exposure. While AgRP population activity shows uniform inhibition, pathway-specific monitoring using axonal GCaMP reveals distinct projection patterns. For example, AgRP terminals in BNST and LH decrease activity to both threat and food, while threats increased AgRP axonal activity in the PVN. Furthermore, location-specific optogenetic inhibition of AgRP neurons conditions spatial avoidance, mimicking a threat-induced defensive behavioural responses. These findings reveal a hypothalamic circuit where DMH GABA neurons suppress AgRP activity in response to external threats, prioritising avoidance over food-seeking to optimise adaptive behavioural responses during foraging.

neuroscience↗

Antibiotic lethality dictates mycobacterial infection outcomes

Antibiotic development and treatment focus on bacterial growth inhibition, often with limited success. Here, we introduce Antimicrobial Single-Cell Testing (ASCT), an advanced imaging strategy to assess bacterial killing in real-time. By tracking 140 million bacteria and generating over 20,000 in vitro time-kill curves, we can predict Mycobacterium tuberculosis treatment outcomes in mice and humans and link strain-specific survival (drug tolerance) in Mycobacterium abscessus to clinical responses. Using ASCT, we reveal drug tolerance as a distinct genetically encoded bacterial trait conserved across drugs with similar targets and, via genome-wide associations, uncover molecular mechanisms that govern bacterial killing. This study establishes the technical framework and in vivo validation for large-scale bacterial killing assessments to advance our understanding of bacterial survival, antibiotic development and clinical decision-making.

microbiology↗