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Shah, N. M.

Publications and source records attributed to Shah, N. M..

4 recordsLinked to original sources

A brain locus for viable gestation

The expectant mother's body is systemically remodeled by her hormones to weather the physiological vicissitudes of pregnancy. In contrast to other reproductive organs, how the expectant mother's brain promotes these system-wide changes is largely unexplored. Here we show that pregnancy induces profound changes in gene expression and identity of hormone-sensitive neurons and report a previously unknown class of such cells that is required for viable pregnancy. We performed RNA sequencing from late mid-gestation mice of four estrogen receptor alpha-expressing populations from hypothalamus and amygdala that regulate reproductive behaviors altered during gestation. Each of these populations undergoes such large, specific transcriptional shifts during pregnancy that these changes even exceed their transcriptional differences between the sexes. The gene expression changes during pregnancy also imbue particular transcriptomically-defined neuronal types within these four populations with new molecular identities. Targeted ablation of one such neuronal type, POANpy2r cells, precludes implantation and abrogates viable pregnancy. Taken together, we have uncovered an essential role of hormone-sensitive neurons in the brain in sustaining pregnancy. The etiology of spontaneous gestational loss, which afflicts ~15% of human pregnancies, remains idiopathic in many cases, and our findings suggest a brain-based mechanism that contributes to such events. More broadly, we provide a molecular and cellular foundation to study gestational processes in health and disease from the perspective of the pregnant brain-body axis.

neuroscience↗

The neural mechanisms supporting the rise and fall of maternal aggression

To protect the helpless young, lactating females dramatically increase aggression towards intruders, known as maternal aggression. However, attack is costly and risky. When pups no longer exist, maternal aggression rapidly declines. Our study reveals the critical role of the pathway from posterior amygdala cells expressing estrogen receptor alpha (PAEsr1) to the ventrolateral part of ventromedial hypothalamus cells expressing neuropeptide Y receptor Y2 (VMHvlNpy2r) in the rise and fall of maternal aggression. Functional manipulations and recordings demonstrate that VMHvl-projecting PAEsr1 (PAEsr1[->]VMHvl) cells are naturally active and required for maternal aggression. During lactation, PA-VMHvlNpy2r connection strengthens, and VMHvlNpy2r excitability increases to facilitate attack. Furthermore, PAEsr1 expresses abundant oxytocin receptors, enabling oxytocin to boost PAEsr1 cell output. After pup separation, the oxytocin level drops, causing decreased maternal aggression, which can be restored by optogenetically increasing the oxytocin level. Thus, diverse forms of plasticity occur at the PAEsr1-VMHvlNpy2r circuit to support need-based maternal aggression.

neuroscience↗

Oxytocin receptor is not required for social attachment in prairie voles

Prairie voles are among a small group of mammals that display long-term social attachment between mating partners. Many pharmacological studies show that signaling via the oxytocin receptor (OxtR) is critical for the display of social monogamy in these animals. We used CRISPR-mutagenesis to independently generate three different OxtR null mutant prairie vole lines. OxtR mutants displayed social attachment such that males and females showed a behavioral preference for their mating partners over a stranger of the opposite sex when assayed using different paradigms. Mothers lacking OxtR delivered viable pups, and parents displayed care of their young and raised them to the weanling stage. Together, our studies unexpectedly reveal that OxtR-mediated signaling is genetically dispensable for social attachment, parturition, and parental behavior.

animal behavior and cognition↗

Evolution of mechanisms that control mating in Drosophila males

Genetically wired neural mechanisms inhibit mating between species because even naive animals rarely mate with other species. These mechanisms can evolve through changes in expression or function of key genes in specific sensory pathways or central circuits. Gr32a is a gustatory chemoreceptor that, in D. melanogaster, is essential to inhibit interspecies courtship and sense quinine. Similar to D. melanogaster, D. simulans Gr32a is expressed in foreleg tarsi, sensorimotor appendages that inhibit interspecies courtship in both species, and it is required to sense quinine. Nevertheless, Gr32a is not required to inhibit interspecies mating by D. simulans males. However, and similar to its function in D. melanogaster, Ppk25, a member of the Pickpocket family, promotes conspecific courtship in D. simulans. Taken together, we have identified shared as well as distinct evolutionary solutions to chemosensory processing of tastants as well as cues that inhibit or promote courtship in two closely related Drosophila species.

neuroscience↗