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Sethuraman, V.

Publications and source records attributed to Sethuraman, V..

2 recordsLinked to original sources

Accumulation of Dietary and Non-Enzymatic Oxysterols in Preeclamptic Placentas: Insights into Cholesterol Dynamics in Pregnancy Complications

Oxysterols--oxidized derivatives of cholesterol--play key roles in regulating lipid metabolism, oxidative stress, and immune signaling. Despite their biological importance, their presence and function in human placental tissue remain poorly understood, particularly in pregnancy complications such as preeclampsia. Preeclampsia is a hypertensive disorder of pregnancy associated with oxidative stress and altered lipid homeostasis. This study profiled both dietary and non-enzymatic oxysterols in third-trimester human placentas from healthy term pregnancies and those complicated with preeclampsia, delivered at term or preterm and term. Findings indicate significantly higher oxysterol levels in preeclamptic placentas, particularly for 7-ketcholesterol (7-Keto) and 5,6{beta}-cholestanetriol, (Triol) regardless of gestational age. 5{beta},6{beta}-epoxycholesterol (OCallaghan, Woods, & OBrien, 2001) was higher in term preeclampsia, while total cholesterol levels were significantly lower in preeclamptic placentas. Correlation analyses revealed distinct associations between oxysterols, cholesterol, and total fat content, indicating unique oxidative signatures in preeclamptic pregnancies. These findings contribute to a growing body of evidence suggesting that altered oxysterol profiles may reflect oxidative stress and placental dysfunction. While causality cannot be inferred, oxysterol profiling may hold potential as a complementary biomarker strategy for identifying pregnancies at risk of adverse outcomes.

physiology↗

Wnt16 exerts pleiotropic effects on bone and lean mass in zebrafish

Bone and muscle interact through developmental, mechanical, paracrine, and autocrine signals. Genetic variants at the CPED1-WNT16 locus are dually associated with bone- and muscle-related traits. While Wnt16 is necessary for bone mass and strength, this fails to explain pleiotropy at this locus. Here, we show wnt16 is required for spine and muscle morphogenesis in zebrafish. In embryos, wnt16 is expressed in dermomyotome and developing notochord, and contributes to larval myotome morphology and notochord elongation. Later, wnt16 is expressed at the ventral midline of the notochord sheath, and contributes to spine mineralization and osteoblast recruitment. Morphological changes in wnt16 mutant larvae are mirrored in adults, indicating that wnt16 impacts bone and muscle morphology throughout the lifespan. Finally, we show that wnt16 is a gene of major effect on lean mass at the CPED1-WNT16 locus. Our findings indicate that Wnt16 is secreted in structures adjacent to developing bone (notochord) and muscle (dermomyotome) where it affects the morphogenesis of each tissue, thereby rendering wnt16 expression into dual effects on bone and muscle morphology. This work expands our understanding of wnt16 in musculoskeletal development and supports the potential for variants to act through WNT16 to influence bone and muscle via parallel morphogenetic processes. AUTHOR SUMMARYIn humans, genetic variants (DNA sequences that vary amongst individuals) have been identified that appear to influence bone and skeletal muscle mass. However, how single genes and genetic variants exert dual influence on both tissues is not well understood. In this study, we found a gene called wnt16 is necessary for specifying the size and shape of muscle and bone during development in zebrafish. Moreover, we disentangled how wnt16 affects both tissues: distinct cellular populations adjacent to muscle and bone secrete Wnt16, where it acts as a signal this guides the size and shape of each tissue. This is important because in humans, genetic variants near the WNT16 gene have effects on both bone- and muscle-related traits. This study expands our understanding of the role of WNT16 in bone and muscle development, and helps to explain how genetic variants near WNT16 affect traits for both tissues. Moreover, WNT16 is actively being explored as a target for osteoporosis therapies; our study could have implications with regard to the potential to target WNT16 to treat bone and muscle simultaneously.

genetics↗