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Petrides, M. J.

Publications and source records attributed to Petrides, M. J..

2 recordsLinked to original sources

A Novel {psi}-χ Fusion Protein for Unravelling the Contributions of χ to DNA Replication and Repair

Faithful DNA replication in Escherichia coli requires the DNA polymerase III holoenzyme (DNA pol III HE) and its clamp loader complex, which couples processive DNA synthesis with {beta}-clamp loading. The clamp loader accessory subunits {chi} and {psi} link single-stranded DNA-binding protein (SSB) to the replisome, stabilizing replication on SSB-coated templates through interactions with the SSB C-terminal tail. Chi has also been implicated in tolerance to the chain-terminating nucleoside analog azidothymidine (AZT), though whether this function depends on {chi} within DNA pol III HE or on its independent interaction with the YoaA helicase remains unclear. To address this, we engineered {psi}-{chi} fusion proteins with flexible glycine-serine linkers to tether the two subunits while preserving folding and activity. Both fusions were biochemically competent, supporting ATP hydrolysis and clamp loading on SSB-coated DNA. In vivo, however, neither the {psi}-GS12-{chi} fusion nor expression of a {psi}{chi} operon restored AZT tolerance in {Delta}holC cells, whereas expression of {chi} alone was sufficient. Fusion expression impaired growth in both wild-type (WT) and {Delta}holC backgrounds, a phenotype alleviated by disrupting {chi}-SSB binding. These findings support a model in which {chi} must dynamically engage SSB and YoaA outside of the clamp loader to promote AZT tolerance, highlighting the importance of regulated {chi}-SSB interactions in genome maintenance.

biochemistry↗

Single-stranded DNA binding protein hitches a ride with the Escherichia coli YoaA-χ helicase

The Escherichia coli XPD/Rad3-like helicase, YoaA, and DNA polymerase III subunit, {chi}, are involved in E. coli DNA damage tolerance and repair. YoaA and {chi} promote tolerance to the DNA chain-terminator, 3{square}-azidothymidine (AZT), and together form the functional helicase complex, YoaA-{chi}. How YoaA-{chi} contributes to DNA damage tolerance is not well understood. E. coli single-stranded DNA binding protein (SSB) accumulates at stalled replication forks, and the SSB-{chi} interaction is required to promote AZT tolerance via an unknown mechanism. YoaA-{chi} and SSB interactions were investigated in vitro to better understand this DNA damage tolerance mechanism, and we discovered YoaA-{chi} and SSB have a functional interaction. SSB confers a substrate-specific effect on the helicase activity of YoaA-{chi}, barely affecting YoaA-{chi} on an overhang DNA substrate but inhibiting YoaA-{chi} on forked DNA. A paralog helicase, DinG, unwinds SSB-bound DNA in a similar manner to YoaA-{chi} on the substrates tested. Through use of ensemble experiments, we believe SSB binds behind YoaA-{chi} relative to the DNA ds/ss junction and show via single-molecule assays that SSB translocates along ssDNA with YoaA-{chi}. This is, to our knowledge, the first demonstration of a mechanoenzyme pulling SSB along ssDNA.

biochemistry↗