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Maruyama, E. O.

Publications and source records attributed to Maruyama, E. O..

3 recordsLinked to original sources

Ascl3+ ionocytes in murine salivary gland ducts are innervated sensory cells that display unique calcium signaling characteristics and contribute to the composition of saliva

Ionocytes are distinct epithelial sensory cells scattered throughout the ductal system of the salivary glands. These cells are distinguished by their unusual morphology, as well as by a specific transcriptomic signature that includes expression of the Foxi1 and Ascl3 transcription factors. Currently, little is known about the biology or function of ionocytes in the salivary glands. To facilitate characterization of these cells, we generated an inducible Cre mouse allele driven by the Ascl3 promoter. This strain was crossed with a reporter to fluorescently label Ascl3+ ionocytes, highlighting that they are the site of enriched CFTR expression in the salivary glands, and demonstrating the proximity of these cells to neurons. Conditional Cre-mediated cell ablation, using diphtheria toxin (DTA), removed Ascl3+ ionocytes from the salivary glands and resulted in an altered pH of total saliva, supporting a function for ionocytes in transepithelial ion flow. Finally, Cre-mediated expression of the calcium indicator GCaMP6f revealed that Ascl3+ ionocytes exhibit unique properties not observed in acinar or surrounding duct cells, including elevated basal [Ca2+]I, spontaneous blinking in the absence of stimulation, and a rapid loss of [Ca2+]I following nerve stimulation. These unique properties distinguish ionocytes as a specialized subset of salivary gland duct cells.

physiology↗

MicroRNA-27a is essential for bone remodeling by modulating p62-mediated osteoclast signaling

The ability to simultaneously modulate a set of genes for lineage-specific development has made microRNA an ideal master regulator for organogenesis. However, most microRNA deletions do not exhibit obvious phenotypic defects possibly due to functional redundancy. MicroRNAs are known to regulate skeletal lineages as the loss of their maturation enzyme Dicer impairs bone remodeling processes. Therefore, it is important to identify specific microRNA essential for bone homeostasis. We report the loss of miR-27a causing severe osteoporosis in mice. MiR-27a affects osteoclast-mediated bone resorption but not osteoblast-mediated bone formation during skeletal remodeling. Gene profiling and bioinformatics further identify the specific targets of miR-27a in osteoclast cells. MiR-27a exerts its effects on osteoclast differentiation through modulation of Squstm1/p62 whose mutations have been linked to Pagets disease of bone. Our findings reveal a new miR-27a-p62 axis necessary and sufficient to mediate osteoclast differentiation and highlight a therapeutic implication for osteoporosis.

developmental biology↗

Short-term and bystander effects of radiation on murine submandibular glands

Many patients treated for head and neck cancers experience salivary gland hypofunction due to radiation damage. Understanding the mechanisms of cellular damage induced by radiation treatment is important in order to design methods of radioprotection. In addition, it is crucial to recognize the indirect effects of IR and the systemic responses that may alter saliva secretion. In this study, radiation was delivered to murine submandibular glands (SMG) bilaterally, using a 137Cs gamma ray irradiator, or unilaterally, using a small animal radiation research platform (SARRP). Analysis at 3, 24 and 48 hours showed dynamic changes in mRNA levels and protein abundance in SMG irradiated bilaterally. Unilateral irradiation using the SARRP caused similar changes in the irradiated SMG, as well as significant off-target, bystander effects in the non-irradiated contralateral SMG. Summary statementRapid changes in gene expression occur in irradiated salivary glands within hours of irradiation, and in non-irradiated contralateral glands, due to bystander effects.

cancer biology↗