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Kowada, R.

Publications and source records attributed to Kowada, R..

2 recordsLinked to original sources

Ribosome stalling facilitates chloroplast targeting of nuclear-encoded proteins

Ribosomes translate mRNAs with variable elongation rates and frequently undergo transient stalling. Although ribosome stalling is known to regulate protein quality control and translational dynamics, its transcriptome-wide landscape and biological significance in plants remain largely unexplored. Here, using disome profiling in Arabidopsis thaliana, we generated a high-resolution map of ribosome stalling sites across the transcriptome and uncovered a marked enrichment on mRNAs encoding chloroplast-targeted proteins, particularly those involved in photosynthesis. We further found that ribosome stalling preferentially occurs when transit peptides emerge from the ribosome exit tunnel. Functional assays demonstrated that deletion of the stalling region significantly reduces chloroplast targeting efficiency. Together, our findings identify ribosome stalling as a regulatory layer that promotes efficient targeting of nuclear-encoded proteins to chloroplasts.

molecular biology↗

An anti-aggregation region of the SGS3 N-terminal IDR is essential for secondary siRNA biogenesis

Secondary siRNA biogenesis amplifies small RNA signals from target transcripts and plays a pivotal role in plant development and defense responses. The RNA-binding protein SGS3 is essential for this pathway, recruiting RNA-dependent RNA polymerase 6 (RDR6) to Argonaute-small RNA-bound targets. The N-terminal intrinsically disordered region (IDR) of SGS3, which contains a prion-like domain (PrLD), has been reported to drive liquid-liquid phase separation, forming siRNA bodies, and to be required for secondary siRNA production. However, the molecular mechanism by which the N-terminal IDR contributes to secondary siRNA production remains unclear. Here, using in vitro reconstitution and in planta assays, we show that the N-terminal IDR comprises two functional modules: the PrLD and a negatively charged region (NCR). The PrLD is required for phase separation and siRNA body formation but is dispensable for secondary siRNA production. In contrast, mutations in the NCR caused SGS3 to form abnormally large cytoplasmic assemblies and markedly impaired secondary siRNA production. These results suggest that the N-terminal IDR helps maintain SGS3 in a functional, soluble state that supports efficient secondary siRNA biogenesis.

molecular biology↗