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Joeloo, M.

Publications and source records attributed to Joeloo, M..

3 recordsLinked to original sources

Detecting CYP2C19 deletions from genotyping array signals using neural networks

Since copy number variations (CNVs) in pharmacogenes can cause significant alterations in drug metabolism, their reliable detection is of high importance both for large-scale studies and personalized medicine. Whole-genome sequencing, and specifically long-read sequencing, is the gold standard for CNV detection. Despite increasing availability of these technologies, genotyping arrays are still widely used as cost-effective alternatives in biobank and clinical settings, yet calling CNVs based on array intensity signals is challenging due to low base pair resolution. In this work, we developed a neural network model, nnCNV, to predict deletions in the CYP2C19 pharmacogene region from array intensity signals. We compared our method to the most widely used algorithm, PennCNV, and demonstrated better performance reaching 100% accuracy in the test dataset. Furthermore, we predicted probe-by-probe CYP2C19 deletion coordinates for all Estonian Biobank samples using nnCNV and PennCNV, and validated these predictions using an identity-by-descent (IBD) sharing method, which also demonstrated superior nnCNV performance. For the deletion samples with conflicting PennCNV and nnCNV predictions, we performed PCR analysis for validation, which showed 97% precision for nnCNV compared to 23% for PennCNV. Finally, we assessed the gradient-based feature importance maps and showed that nnCNV utilizes signal intensity information not only from deletion probes, but also from probes in flanking regions. Our results demonstrate that long-range information, which cannot be utilized by hidden Markov models, can improve CNV calling.

bioinformatics↗

StarPhase: Comprehensive Phase-Aware Pharmacogenomic Diplotyper for Long-Read Sequencing Data

Pharmacogenomics is central to precision medicine, informing medication safety and efficacy. Phar-macogenomic diplotyping of complex genes requires full-length DNA sequences and detection of structural rearrangements. We introduce StarPhase, a tool that leverages PacBio HiFi sequence data to diplotype 21 CPIC Level A pharmacogenes and provides detailed haplotypes and supporting visualizations for HLA-A, HLA-B, and CYP2D6. StarPhase diplotypes have high concordance with benchmarks where 99.5% are either exact matches or minor discrepancies. Manual inspection of the 0.5% mismatches indicates they were correctly called by StarPhase. With StarPhase, we update or correct 26.2% of GeT-RM pharmacogenomic diplotypes. Population distributions from StarPhase mostly reflect those of the All of Us cohort, while also highlighting gaps in existing pharmacogenomic databases that long-read sequencing can fill. With a single HiFi whole genome sequencing assay, StarPhase enables robust PGx diplotyping even as additional pharmacogenes and haplotypes are discovered.

bioinformatics↗

Genetic determinants of plasma protein levels in the Estonian population

The proteome holds great potential as an intermediate layer between the genome and phenome. Previous protein quantitative trait locus studies have focused mainly on describing the effects of common genetic variations on the proteome. Here, we assessed the impact of the common and rare genetic variations as well as the copy number variants (CNVs) on 326 plasma proteins measured in up to 500 individuals. We identified 184 cis and 94 trans signals for 157 protein traits, which were further fine-mapped to credible sets for 101 cis and 87 trans signals for 151 proteins. Rare genetic variation contributed to the levels of 7 proteins, with 5 cis and 14 trans associations. CNVs were associated with the levels of 11 proteins (7 cis and 5 trans), examples including a 3q12.1 deletion acting as a hub for multiple trans associations; and a CNV overlapping NAIP, a sensor component of the NAIP-NLRC4 inflammasome which is affecting pro-inflammatory cytokine interleukin 18 levels. In summary, this work presents a comprehensive resource of genetic variation affecting the plasma protein levels and provides the interpretation of identified effects.

genomics↗