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Jaryani, F.

Publications and source records attributed to Jaryani, F..

3 recordsLinked to original sources

MosaicSim: A Novel Mosaic Variant Simulator Reveals Diminishing Returns of Ultra-High Coverage for Mosaic Variant Detection

Genetic mutations within select cells of a tissue, termed mosaic variants (MV), are being increasingly recognized for their role in human disease. This growing interest underscores the need for specialized tools to detect and analyze MVs. However, such detection methods still lack thorough evaluation, largely due to missing benchmarking datasets that are large, reliable, and reflective of the complexity of biological samples. To address this gap, we developed MosaicSim, a tool for simulating variants in realistic sequencing data. The TweakVar workflow is at the tools core and represents a unique simulation pipeline that layers simulated MVs onto empirical whole genome sequencing data, generating a large, realistic ground truth dataset that combines the strengths of both simulation and biological data. To demonstrate the functionality of the workflow, we simulated 1,000 mosaic single nucleotide polymorphisms using TweakVar within whole genome sequencing files of different coverages. MVs were called with Illuminas DRAGEN and compared to the ground truth. Our results show 150x-445x coverage performed comparably, with a true-positive rate between 50.4% (300x) and 54.9% (150x) and no false-positives detected. Across all samples, increasing variant allele frequency had a significant positive effect on call success. Additionally, we observed that call rates for variants in lower complexity regions improved with increasing read depth. We did not find significant effects attributable to specific mutation patterns or mean read map quality. MosaicSim fills a critical unmet need by providing representative, customizable ground truth datasets for MV benchmarking, enabling systematic evaluation and optimization of variant calling methods.

genomics↗

EZHIP boosts neuronal-like synaptic gene programs and depresses polyamine metabolism

It is currently understood that the characteristic loss of the repressive histone mark H3K27me3 in PFA ependymoma and diffuse midline glioma (DMG) are caused by complementary mechanisms mediated by EZHIP and the oncohistone H3K27M, respectively. To support the complementarity of these mechanisms, rare H3K27M-negative DMGs express EZHIP. Interestingly, EZHIP is one of the few genes recurrently mutated in PFA. The significance of EZHIP mutations in PFA, and whether EZHIP has wider functions in addition to repression of H3K27me3 deposition, are not known. Here, we investigated the mutational landscape of EZHIP in pediatric brain tumors. We found that EZHIP mutations occur not only in PFA, but also in rare medulloblastoma and pediatric high-grade glioma (HGG), including in H3K27-positive DMG. Contrary to current expectations, we show that mutant EZHIP is expressed in H3K27M-positive DMG. All the EZHIP-mutated HGG cases also have EGFR mutations. Further, we pursued better understanding of the function of EZHIP by expressing it in human-derived neural models. Our transcriptomic analyses indicate that EZHIP expression potentiates neuronal-like gene programs associated with synaptic function. Metabolomics data indicate that EZHIP leads to repression of methionine and polyamine metabolism, suggesting links between metabolic and epigenetic changes that are observed in PFA. Collectively, our results expand the repertoire of tumor types known to harbor EZHIP mutations and shed light on EZHIP-dependent metabolic and transcriptional programs in relevant neural models.

cancer biology↗

DesiRNA: structure-based design of RNA sequences with a Monte Carlo approach

RNA sequences underpin the formation of complex and diverse structures, subsequently governing their respective functional properties. Despite the pivotal role RNA sequences play in cellular mechanisms, creating optimized sequences that can predictably fold into desired structures remains a significant challenge. We have developed DesiRNA, a versatile Python-based software tool for RNA sequence design. This program considers a comprehensive array of constraints, ranging from secondary structures (including pseudoknots) and GC content, to the distribution of dinucleotides emulating natural RNAs. Additionally, it factors in the presence or absence of specific sequence motifs and prevents or promotes oligomerization, thereby ensuring a robust and flexible design process. DesiRNA utilizes the Monte Carlo algorithm for the selection and acceptance of mutation sites. In tests on the EteRNA benchmark, DesiRNA displayed high accuracy and computational efficiency, outperforming most existing RNA design programs.

bioinformatics↗