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Haefner, V.

Publications and source records attributed to Haefner, V..

3 recordsLinked to original sources

Establishing toxicologically relevant primary alveolar macrophage models to enable a realistic assessment of nanomaterials safety

Alveolar macrophages (AMs) are key sentinels of the distal lung, maintaining surfactant homeostasis and clearing inhaled particles through continuous surveillance and phagocytosis. Mechanistic studies of particle-cell interactions, important to assess the respiratory safety or toxicity of particles, however, are limited by the lack of suitable in vitro models that preserve the core features of resident AMs. Here, we benchmarked six murine AM models, including short-term cultured primary AMs (AMs 3 h, AMs 24 h), primary AMs maintained with GM-CSF, TGF-{beta}, and the PPAR-{gamma} agonist rosiglitazone (AMs GTR), bone marrow-derived AM-like cells (BM-AMs), the immortalized AM cell line MH-S, and the non-transformed fetal liver-derived AM-like cell line MPI, against in vivo AMs based on surface phenotype, transcriptomic identity, and their responses to three carbon-based nanomaterials (CBNs). Both cell lines showed limited AM identity, characterized by low CD11c and SiglecF but high CD11b surface expression. In contrast, AMs GTR and BM-AMs more closely preserved resident AM-like surface marker profiles. Transcriptomic features, although differing for alterations in metabolism-related pathways, particularly oxidative phosphorylation, revealed most in vivo-like for the primary AM models. Functional, toxicological assessment using soot-like carbon nanoparticles (CNP), double-walled carbon nanotubes (DWCNT) and multi-walled carbon nanotubes (MWCNT) demonstrated strong model-dependent responses. Consistent with MWCNT-induced reduction in AM numbers observed in vivo, AMs GTR and BM-AMs exhibited the strongest in vitro responses, including cytotoxicity and inflammatory cytokine release. MH-S cells in contrast stood out for their cytotoxic sensitivity supporting their use for high throughput cytotoxicity tests. Overall, these findings identify AMs GTR and BM-AMs as the closest models for studying AMs biology and AM-intrinsic responses to poorly soluble nanoparticles like CBNs, and with regard to availability of higher cell numbers, BM-AMs might provide a scalable platform for mechanistic and higher-throughput nanotoxicology studies.

pharmacology and toxicology↗

Cell circuits underlying nanomaterial specific respiratory toxicology

Nanomaterials emerged as boundless resource of innovation, but their shape and biopersistence related to respiratory toxicology raise longstanding concerns. The development of predictive safety tests for inhaled nanomaterials, however, is hampered by limited understanding of cell type-specific responses. To advance this knowledge, we used single-cell RNA-sequencing to longitudinally analyze cellular perturbations in mice, caused by three carbonaceous nanomaterials of different shape and toxicity upon pulmonary delivery. Focusing on nanomaterial-specific dynamics of lung inflammation, we found persistent depletion of alveolar macrophages by fiber-shaped nanotubes. While only little involvement was observed for alveolar macrophages during the initiation phase, they emerged, together with infiltrating monocyte-derived macrophages, as decisive factors in shifting inflammation towards resolution for spherical nanomaterials, or chronic inflammation for fibers. Fibroblasts, central for fibrosis, sensed macrophage and epithelial signals and emerged as orchestrators of nanomaterial-induced inflammation. Thus, the mode of actions identified in this study will significantly inspire the precision of future in vitro testing. Graphical abstract O_FIG O_LINKSMALLFIG WIDTH=200 HEIGHT=80 SRC="FIGDIR/small/579746v1_ufig1.gif" ALT="Figure 1"> View larger version (31K): org.highwire.dtl.DTLVardef@1d6f061org.highwire.dtl.DTLVardef@3fabb1org.highwire.dtl.DTLVardef@21040eorg.highwire.dtl.DTLVardef@1915c3b_HPS_FORMAT_FIGEXP M_FIG C_FIG

pharmacology and toxicology↗

Tobacco TGA7 mediates gene expression dependent and independent of salicylic acid

Basic region leucine zipper (bZIP) transcription factors of the TGA family control gene expression in response to diverse stimuli. Arabidopsis clade II and clade III TGA factors mediate salicylic acid (SA)-induced expression of PATHOGENESIS-RELATED GENE1 (PR-1) via interplay with NONEXPRESSOR OF PR GENES1 (NPR1, a.k.a. NIM1). Interaction with TGA factors occurs through the central ankyrin repeat domain of NPR1. In a yeast two-hybrid screen with the NPR1 bait, we identified TGA7, a novel member of the tobacco (Nt) TGA family grouping to clade III. TGA7 is most similar to NtTGA1a, and, like NtTGA1a, TGA7 displays transcription activity in yeast. Unexpectedly, TGA7 preferentially and uniquely interacts with the SA-sensitive C-terminal region of NtNPR1, demonstrating that NtNPR1 harbors multiple distinct TGA factor binding sites. Interaction with NPR1 impairs TGA7 transcription activity in yeast. Furthermore, TGA7 binding to the NtNPR1 C-terminus is outcompeted by SA-induced type 2 NIM1-INTERACTING (NIMIN) proteins. In tobacco plants, a TGA7-Gal4 DNA-binding domain chimeric protein (TGA7GBD) mediates SA-responsive reporter gene expression in young leaf tissue and spontaneous reporter activation in older leaves displaying PR-1 gene expression. Astonishingly, TGA7GBD is also able to activate the reporter independent from PR-1 gene expression in noninduced cotyledons of tobacco seedlings. Together, our findings support a model in which TGA7 mediates both SA-dependent and SA-independent gene activation controlled by the plants developmental stage and by the C-terminal region of constitutively accumulating NtNPR1.

plant biology↗