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Biology subjects

De Vita, G.

Publications and source records attributed to De Vita, G..

3 recordsLinked to original sources

AI-Driven Computational Design of Peptide-Based WWP1 Inhibitors as Promising Therapeutic Agents Against Breast Cancer, Including Triple-Negative Subtype

Breast cancer (BC) is the second most common noncutaneous cancer and the second leading cause of cancer-related death in women. BC is classified into three primary subtypes, with triple-negative breast cancer (TNBC) having the poorest prognosis because it lacks specific targetable markers. Preclinical studies on TNBC indicated a common occurrence of diminished tumor-suppressor activity of PTEN, activating the PI3K/AKT/mTOR signaling pathway. Notably, published studies reveal that the WWP1 enzyme plays a pivotal role in driving PTEN degradation via ubiquitination, unveiling a promising therapeutic target for treating TNBC. In the search of new WWP1 inhibitors, we used artificial intelligence (AI)-driven computational strategies for de novo design of peptide-based WWP1 inhibitors and identified a hexapeptide, termed WI23-B, which demonstrated high nanomolar binding affinity to WWP1. In TR-FRET enzymatic assays, WI23-B inhibited WWP1 activity with an IC of approximately 11 {micro}M. In MCF7 and MDA-MB-231 breast cancer cell lines, WI23-B showed promising cytotoxic efficacy, particularly in combination with the PI3K inhibitor BYL719, also when it was loaded into nanocapsules. Collectively, these findings highlight WI23-B as a promising lead peptide with potent WWP1 inhibitory activity and synergistic antiproliferative effects when combined with PI3K inhibitors. While further structural optimization is required to enhance its potency and pharmacological properties, our results provide a strong foundation for the development of next-generation WWP1 inhibitors. Such agents have the potential to reshape therapeutic strategies for BC and TNBC by enabling more effective and less toxic treatment regimens, ultimately reducing the reliance on high-dose chemotherapy and minimizing adverse effects.

cancer biology↗

Radiation-induced interferon-I response impairs thyroid organoid function

Background and AimRadiotherapy is a standard cancer treatment, but radiation exposure to surrounding healthy tissues may lead to adverse side effects that compromise patient quality of life. In patients with head and neck cancer treated with radiotherapy, thyroid damage is a frequent complication, resulting in hypothyroidism or secondary thyroid malignancies. Although clinically recognized, the molecular mechanisms underlying these side effects remain mostly unexplored. This study aims to characterize the radiation-induced molecular alterations in thyroid organoids. MethodsBulk RNA sequencing was performed to investigate transcriptomic changes in tissue-derived thyroid organoids following gamma-irradiation. Observed changes were further validated and explored using qPCRs, western blotting, immunofluorescence, caspase 3/7 activity and organoid forming efficiency. ResultsOur findings identify interferon-{beta} (IFN-{beta}) signaling as a key mediator of radiation-induced inflammation in the thyroid. Additionally, the intrinsic apoptotic pathway was found to be the predominant mechanism of radiation-induced thyroid cell death. Notably, while IFN-{beta} exhibited a protective effect against apoptosis, it concurrently reduced thyroid stem progenitor cell potential. ConclusionsThese results highlight the dual role of IFN-{beta} signaling in modulating thyroid cell fate after irradiation, potentially promoting survival upon injury at the expense of regenerative potential.

cell biology↗

Role of Klhl14 in senescence and epithelial-to-mesenchymal transition via TGF- modulation

KLHL14, a component of an E3-ubiquitin ligase complex, has emerged as a context-dependent oncogene or tumor suppressor, particularly important for thyroid development. Yet its role in thyroid biology remains largely unexplored. In this study, we uncover a central function for KLHL14 in maintaining thyroid epithelial identity and regulating tissue homeostasis. Using a thyroid organoid model, we show that KLHL14 is essential for the proper growth and maturation of thyroid cells. Reduction of KLHL14 expression disrupts organoid development and triggers a dual cellular response involving features of both senescence and epithelial-to-mesenchymal transition. These phenotypic changes are accompanied by increased cellular plasticity and migratory capacity. Mechanistically, we identify TGF-{beta} signaling as a key pathway activated upon KLHL14 depletion, contributing to the observed cellular reprogramming. Inhibiting TGF-{beta} restores growth and reduces markers of senescence and EMT, positioning KLHL14 as an upstream modulator of this signaling axis. These findings reveal a previously unrecognized role for KLHL14, suggesting that its dysfunction may contribute to disease progression in aggressive thyroid cancers. This work broadens our understanding of thyroid epithelial biology and provides molecular insights extendable to other tissues, highlighting KLHL14 as a potential target for therapeutic interventions in malignancies displaying the herein explored features.

cell biology↗