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Dahan, E.

Publications and source records attributed to Dahan, E..

3 recordsLinked to original sources

DNT: Diploid Genomic Foundation Model

Clinical interpretation of genetic variation depends on the diploid genotype, including zygosity, allele dosage and whether multiple variants occur in cis on the same homologue or in trans on different homologues. Most genomic language models process haploid sequences or combine independently encoded haplotypes downstream, so they do not directly represent the paired genotype in a single sequence. We introduce a reference-aligned diploid encoding for single-nucleotide variants (SNVs) and short insertions and deletions (indels), together with unphased and phase-retaining tokenizers that accept phased genotypes and convert them to single-sequence diploid representation. Using Nucleotide Transformer v3 backbones, we continue training 8-million- and 100-million-parameter models and evaluate an auxiliary Contrastive Phase Loss (CPL) designed to retain the phasing information of the variants in contextual representations. We evaluate on a novel compound-heterozygous benchmark containing 9,460 examples. Models whose inputs did not distinguish relative phase remained near chance, whereas our diploidic models improved discrimination with AUROC 0.649, compared to 0.506 for the vocabulary-adapted control. These findings establish a method for making diploid genotype information accessible to genomic language models, rather than a universal improvement in variant prediction; validation in naturally observed, accurately phased clinical cohorts remains necessary.

bioinformatics↗

GFMBench-API: A Standardized Interface for Benchmarking Genomic Foundation Models

The rapid scaling of Genomic Foundation Models (GFMs) has created a critical need for standardized evaluation frameworks. Current benchmarking practices are often fragmented, relying on model-specific preprocessing and inconsistent metric implementations that hinder reproducible comparisons. We present GFMBench-API, a high-level Python interface designed to unify the evaluation lifecycle of GFMs. GFMBench-API provides a modular "middleware" architecture that decouples model-specific tokenization and embedding logic from task-specific data streams and performance metrics. By standardizing the input/output schemas for common genomic tasks, such as regulatory element prediction, variant effect scoring, and long-range interaction mapping, GFMBench-API enables researchers to integrate new models or tasks with minimal "glue code." Our interface ensures mathematical consistency across evaluations, providing a robust foundation for the transparent and systematic benchmarking of GFMs.

genomics↗

Poly ADP-ribosylation regulates Arc expression and promotes adaptive stress-coping

Rapid adaptation to stressful events is essential for survival and requires acute stress response and stress-coping strategy. Yet, the molecular mechanisms that govern this coping strategy have to be fully discovered. Poly ADP-ribosylation (PARylation) is a post-translational protein modification that plays a vital role in histone modification following neuronal activity and is required for long-term memory formation. Previous studies showed that inhibiting Parp1, the primary enzyme that catalyzes poly ADP-ribose polymers (PAR), impairs long-term memory formation. However, the target genes of Parp1-induced PARylation following acute stress are unknown. Here, we showed that the forced swim test, a well-established acute stress paradigm, induced elevated cortical PARylation and increased the expression of activity-dependent genes in mice. Systemic pharmacological Parp1 inhibition with ABT888 (Veliparib), impaired stress-coping in a second forced swim test done 24 hours later as measured by diminished immobility response. This effect was associated with reduced PARylation of Arc promoter and reduced Arc expression, 1 hour after Parp1 inhibition, suggesting Arc PARylation regulates its expression and hence successful stress-coping behavior.

neuroscience↗