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Bloch, V.

Publications and source records attributed to Bloch, V..

2 recordsLinked to original sources

CNN and transfer learning-based classification model for automated cows feeding behaviour recognition from accelerometer data

Due to technological developments, wearable sensors for monitoring farm animal behaviour have become cheaper, with longer life and more accessible for small farms and researchers. In this study, an acceleration measuring tag connected by BLE for monitoring behaviour of dairy cows was used. An optimal CNN-based model for the feeding behaviour classification was trained and the training process was analysed considering training dataset and the use of transfer learning. A classifier based on a neural network was trained by acceleration data collected in a research barn. Based on a dataset including 33.7 cow*days (21 cow recorded during 1-3 days) of labelled data and an additional free access dataset with similar acceleration data, a classifier with F1=93.9% was developed. The optimal classification window size was 90s. In addition, the influence of the training dataset size on the classifier accuracy was analysed for different neural networks using the transfer learning technique. During increasing of the training dataset size, the rate of the accuracy improvement decreased, and, starting from a specific point, the use of additional training data can be impractical. Relatively high accuracy was achieved with few training data when the classifier was trained using randomly initialised model weights, and higher accuracy was achieved when transfer learning was used. These findings can be used for estimation of the necessary dataset size for training neural network classifiers intended for other environments and conditions. HighlightsCNN cow feeding behaviour classifier was optimised for neck tags. Transfer learning technique significantly improves accuracy of CNN. Relation between the training dataset size and CNN accuracy was estimated. NN trained by one dataset can be inapplicable for others. BLE tags measuring acceleration transferred data in real time.

animal behavior and cognition↗

Gene-Gene interactions and pleiotropy in the brain nicotinic pathway associated with the heaviness and precocity of tobacco smoking among outpatients with multiple substance use disorders

IntroductionTobacco smoking is a major health burden worldwide, especially in populations suffering from other substance use disorders (SUDs). Several smoking phenotypes have been associated with single nucleotide polymorphisms (SNPs) of nicotinic acetylcholine receptors (nAChRs). Yet, little is known about the genetics of tobacco smoking in populations with other SUDs, particularly regarding gene-gene interactions and pleiotropy, which are likely involved in the polygenic architecture of SUDs. Thus, we undertook a candidate pathway association study of nAChR-related genes and smoking phenotypes in a sample of SUD patients.\n\nMethods493 patients with genetically-verified Caucasian ancestry were characterized extensively regarding patterns of tobacco smoking, other SUDs, and 83 SNPs from the nicotinic pathway, encompassing all brain nAChR subunits and metabolic/chaperone/trafficking proteins. Single-SNP, gene-based and SNP x SNP interactions analyses were performed to investigate associations with relevant tobacco smoking phenotypes. This included Bayesian analyses to detect pleiotropy, and adjustment on clinical and sociodemographic confounders.\n\nResultsAfter multiple adjustment, we found independent associations between CHRNA3 rs8040868 and a higher number of cigarettes per day (CPD), and between RIC3 rs11826236 and a lower age at smoking initiation. Two SNP x SNP interactions were associated with age at onset (AAO) of daily smoking. There was pleiotropy regarding three SNPs in CHRNA3 (CPD, AAO daily smoking), ACHE (CPD, HSI) and CHRNB4 (CPD, both AAOs).\n\nDiscussionDespite limitations, the present study shows that the genetics of tobacco smoking in SUD patients are both distinct and partially shared across smoking phenotypes, and involve metabolic and chaperone effectors of the nicotinic pathway.

genetics↗