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Aamodt, K. I.

Publications and source records attributed to Aamodt, K. I..

2 recordsLinked to original sources

Mapping histological and functional maturation of human endocrine pancreas across early postnatal periods

Human endocrine cell differentiation and islet morphogenesis play critical roles in determining islet cell mass and function, but the events and timeline of these processes are incompletely defined. To better understand early human islet cell development and maturation, we collected 115 pediatric pancreata and mapped morphological and spatiotemporal changes from birth through the first ten years of life. Using quantitative analyses and a combination of complementary tissue imaging approaches, including confocal microscopy and whole-slide imaging, we developed an integrated model for endocrine cell formation and islet architecture, including endocrine cell type heterogeneity and abundance, endocrine cell proliferation, and islet vascularization and innervation. We also assessed insulin and glucagon secretory profiles in isolated islet preparations from pediatric donors aged 2 months to 10 years and found a temporal difference in the maturation of insulin secretion compared to glucagon secretion. This comprehensive summary of postnatal and pediatric pancreatic islet development provides a framework for future studies and integration of emerging genetic and genomic data related to islet biology and diabetes risk.

developmental biology↗

Coordinated interactions between endothelial cells and macrophages in the islet microenvironment promote beta cell regeneration

Endogenous {beta} cell regeneration could alleviate diabetes, but proliferative stimuli within the islet microenvironment are incompletely understood. We previously found that {beta} cell recovery following hypervascularization-induced {beta} cell loss involves interactions with endothelial cells (ECs) and macrophages (M{Phi}s). Here we show that proliferative ECs modulate M{Phi} infiltration and phenotype during {beta} cell loss, and recruited M{Phi}s are essential for {beta} cell recovery. Furthermore, VEGFR2 inactivation in quiescent ECs accelerates islet vascular regression during {beta} cell recovery and leads to increased {beta} cell proliferation without changes in M{Phi} phenotype or number. Transcriptome analysis of {beta} cells, ECs, and M{Phi}s reveals that {beta} cell proliferation coincides with elevated expression of extracellular matrix remodeling molecules and growth factors likely driving activation of proliferative signaling pathways in {beta} cells. Collectively, these findings suggest a new {beta} cell regeneration paradigm whereby coordinated interactions between intra-islet M{Phi}s, ECs, and extracellular matrix mediate {beta} cell self-renewal.

cell biology↗